CAOPORN成人免费公开,欧美FREESEX黑人又粗又大 ,欧美狠狠入鲁的视频777色,主仆调教SM束缚绳索捆绑

掃碼關(guān)注公眾號           掃碼咨詢技術(shù)支持           掃碼咨詢技術(shù)服務(wù)
  
客服熱線:400-901-9800  客服QQ:4009019800  技術(shù)答疑  技術(shù)支持  質(zhì)量反饋  人才招聘  關(guān)于我們  聯(lián)系我們
好爽又高潮了毛片免费下载,甜蜜的妻子,无码毛片AAA在线
Human ox-LDL (bs-1698P)  
訂購熱線:400-901-9800
訂購郵箱:sales@www.tjshangbang.com
訂購QQ:  400-901-9800
技術(shù)支持:techsupport@www.tjshangbang.com
說明書: 1mg  2mg  
1mg/2600.00元
2mg/4000.00元
大包裝/詢價
產(chǎn)品編號 bs-1698P
英文名稱 Human ox-LDL
中文名稱 氧化低密度脂蛋白
別    名 ox-LDL: LDL (Copper oxidized); Cu2SO4 oxidized low density lipoprotein; oxidized low density lipoprotein; Low density lipoprotein; MDA oxidized LDL; MDA oxidized low density lipoprotein; Human ox-LDL  氧化型低密度脂蛋白;
Specific References  (7)     |     bs-1698P has been referenced in 7 publications.
[IF=6.986] Yang, Yilin. et al. Knockdown of lncRNA H19 alleviates ox-LDL-induced HCAECs inflammation and injury by mediating miR-20a-5p/HDAC4 axis. INFLAMM RES. 2022 Jul;:1-13  
[IF=3.647] Yi Zhou et al. Long non-coding RNA MIAT/miR-148b/PAPPA axis modifies cell proliferation and migration in ox-LDL-induced Human aorta vascular smooth muscle cells. Life Sci. 2020 Sep 1;256:117852.  Other ;  Human.  
[IF=3.514] Su Q et al. Pinocembrin protects endothelial cells from oxidized LDL-induced injury.Cytokine. 2018 Nov;111:475-480.  Other ;  
[IF=3.466] Zhang et al. H19 knockdown suppresses proliferation and induces apoptosis by regulating miR-148b/WNT/β-catenin in ox-LDL -stimulated vascular smooth muscle cells. (2018) J.Biomed.Sci. 25:11  other ;  Human.  
[IF=3.309] Ye J et al. LncRBA GSA5, up-regulated by ox-LDL, aggravates inflammatory response and MMP expression in THP-1 macrophages by acting like a sponge for miR-221.Exp Cell Res. 2018 Aug 15;369(2):348-355.  Other ;  
[IF=2.1] Heng-Jing Hu. et al. Hydrogen sulfide improves ox?LDL?induced expression levels of Lp?PLA2 in THP?1 monocytes via the p38MAPK pathway. Mol Med Rep. 2021 May;23(5):1-10  Other ;  
[IF=1.785] Huiqiong Jiang. et al. MicroRNA?301a?3p increases oxidative stress, inflammation and apoptosis in ox?LDL?induced HUVECs by targeting KLF7. Exp Ther Med. 2021 Jun;21(6):1-8  WB,FC ;  Human.  
性    狀 Liquid
濃    度 >1.0 mg/ml
物    種 Human
序    列 Purified native protein
內(nèi)毒素 Not analyzed
活性 Not tested
緩 沖 液 10 mM PBS(pH=7.4) with 0.01 μM EDTA.
保存條件 Store at 4°C for 8-20 weeks. Avoid freeze.
注意事項 This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.
產(chǎn)品介紹 Low-density lipoprotein (LDL) is the carrier protein for cholesterol in the blood. LDL binds to its receptor on the capillary walls and thereby mediates the uptake and clearence of cholesterol from the circulation. In atherosclerotic lesions oxidatively modified LDL is found and oxidized LDL is specifically recognized and ingested by macrophages via scavenger receptor A and CD36. Oxidized LDL may be a marker of atherosclerosis but the precise changes in oxidized LDL are not well described. Low-density lipoprotein oxidised with Cu2SO4.

SWISS:
N/A

Gene ID:
N/A

來源:正常人血漿中分離獲得,經(jīng)銅離子氧化人源血漿低密度脂蛋白;。
純度:98%,(瓊脂糖凝膠電泳)
注意事項:
1. 長時間的儲存后可能會產(chǎn)生沉淀,這種現(xiàn)象屬于正常情況。將溶液放入離心管中離心2分鐘將聚合物除去即可。
2. 高密度脂蛋白相當(dāng)不穩(wěn)定,提前計劃你的實驗,使用新鮮的材料,建議實驗時最好現(xiàn)用現(xiàn)配,采用新鮮配置工作液。
版權(quán)所有 2004-2026 www.www.tjshangbang.com 北京博奧森生物技術(shù)有限公司
通過國際質(zhì)量管理體系ISO 9001:2015 GB/T 19001-2016    證書編號: 00124Q34771R2M/1100
通過國際醫(yī)療器械-質(zhì)量管理體系ISO 13485:2016 GB/T 42061-2022    證書編號: CQC24QY10047R0M/1100
京ICP備05066980號-1         京公網(wǎng)安備110107000727號
国产大片黄在线观看私人影院| 亚洲精品aaaa久久久久久| 最爽无遮挡行房视频| 精品日韩一区二区三区| 91高清欧美黄色| 24小时日本在线WWW免费的| 国产在线免费视频观看| 亚洲色婷婷一区二区三区| 一区2区3区精品国产欧美| 女同一区二区在线观看| 两个女人互添下身爽舒服小说| 国内久久| 香蕉在线精品视频在线观看2| 国内精品久久久久久久久野战| 人妻激情另类乱人伦人妻| 精品国自产拍天天拍| 精品福利一区二区三区免费视频| 亚洲AV综合色区无码一二三区| 欧美日韩在线激情| 国产成本人片免费AV| 国产综合在线播放| 亚洲精品美女久久久久9999| 视频一区视频二区视频三区80| 亚洲国产成人久久综合一区77| 日韩欧美亚洲国产精品字幕久久久| 中英字幕乱码在线观看| 国产AV无码专区亚洲A∨毛片| 东京热男人aV天堂| 精品多毛少妇人妻AV免费久久| 日本高清在线精品一区二区三区| 欧美丰满少妇XXXXX| 日韩欧美中文字幕在线四区| 国产在线观看黄AV免费| 丰满少妇张开大白腿摄影图片| 欧美激情第1页| 不卡一区二区三区卡| 耻辱の中出し授业大桥未久| 无翼乌之全彩爆乳口工不知火舞| 中文字幕日韩精品有码视频| 国产精品久久久久AV| 中文字幕av久久|